INTRODUCTIONDefective conversion of 17-hydroxyprogesterone (17OHP) to 11-deoxycortisol accounts for more than 95 percent of cases of congenital adrenal hyperplasia (CAH) [1,2]. This conversion is mediated by 21-hydroxylase due to mutations in the CYP21A2 gene. Based upon neonatal screening studies that detect classic CAH, 21-hydroxylase deficiency (21OHD) is one of the more common inherited disorders. The […]
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